[Recovered File]
Protein STX-Delta9 and the Limits of Human-Mycelial Symbiosis:
Genomic Integration of Hyperadaptive Mycotic Sequences
Classified Level: IV
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Under directive #####, Project CHAOS-7 was initiated to explore the mutagenic properties of Neocordyceps xenoformis, a hyper-adaptive fungal strain discovered in the ###### Exclusion Zone. Primary objectives included isolation, integration of genetic factors responsible for resilience, recreation of specific neuroplasticity triggered in infected specimens and examinations of regenerative capabilities into Homo sapiens. The study was conducted over several weeks using CRIPSR-Cas9 gene editing and strict monitoring of specific markers and proteins.
Initial hypotheses suggested that N. xenoformis contained a sequence of anomalous proteins capable of extending metabolic activity in hypoxic conditions, enhancing neural synaptic plasticity, and triggering controlled morphogenesis in host tissue. The directive aimed to reveal the presence of the protein and utilise its properties in potentially limitless applications. They include: improving living conditions, controlling evolution in extreme environments, and increasing combat resilience and resistance to N. xenoformis. Throughout this study, we showed the presence of the protein in our test subject throughout the infection cycle and analysed the inferred biological reaction.
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Specimen #1421 originated from Subject H-9, a male volunteer selected for his exceptional physiological markers. Standard CRISPR vector injections were applied, replacing critical genome sequences for the N. xenoformis strand.
Initial cellular responses were promising: enhanced ATP synthesis, accelerated mitosis in injured organs and delayed resistance to apoptosis. Deviations from expected parameters occurred at T+72 hours when neuronal clusters in the subject’s basal ganglia began exhibiting autonomous activity, leading to involuntary motor functions.
By T+96 hours, leukocyte activity had collapsed, triggering extensive tissue necrosis followed by unanticipated and uncontrolled tissue regrowth and lymph production. Subject H-9 ceased all verbal communication at this stage, responding only to external stimuli with erratic behaviours. Neural scans revealed excessive dendritic branching and inter-cranial pressure spikes indicative of uncontrolled cerebral expansion.
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By T+120 hours, Subject H-9 was designated Specimen #1421. Deviations from human physiology were advanced, and conventional means of sedation inconclusive. We ordered and advised for continuous containment adjustments.
By T+168 hours, Specimen #1421 displayed several new biological anomalies:
- Rapid muscle fibre regeneration
- Reduced ability to succumb to conventional organ failure
- Sustained neuromuscular expansion
- Chaotic formation of supplementary appendages exhibiting autonomous movement (1)
- Heightened aggression in response to biological stimuli, particularly hunger and fear
(1) Upon examining the genome sequence of said appendages, we later revealed more than 98% similarity with N. xeroformis as well as mirrored behaviours, including controlled expansions, creation of tendril-like structures, and release of reproducing agents.
Containment protocol was elevated, ensuing unsuccessful sterilisation protocol and termination orders were drafted. During scheduled incineration, Specimen #1421 exhibited unpredicted limb detachment behaviours as well as accelerated sclerosis. Carbon-based combustive measures failed to halt propagation due to mycelial sprouting in fragmented tissues.
Biohazard teams initiated Lockdown Protocol 7.
Containment breach occurred at T+216 hours, showcasing impressive astute thinking and potential planning capacity. ## casualties were later confirmed in an attempt to recapture and relocate the subject.
Specimen #1421 was relocated to Site-## for indefinite containment. Further experimentation suspended indefinitely.
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While Specimen #1421 remains nonviable as a controlled subject, its physiological development offers key insights for future research, as outlined in this research paper. Throughout three parts, we’ll provide detailed protocols, stages of infection and analysis of acquired data.
Several recommendations are formulated and summarised here:
1. Observations of Protein STX-Delta9: Enzyme activity correlates with accelerated mycelial proliferation and neural activity. The isolation of the protein and refinement of CRISPR vector delivery may allow for a controlled evaluation without total cognitive collapse.
2. Targeted Neural Adaptation: Cerebral activity resulted in loss of coherent function. Implementing staged mutations across cortical regions may prevent uncontrolled synaptic growth.
3. Suppressor Gene Integration: A failsafe suppression mechanism should be engineered to counteract excessive host-fungal integration, and another study is being conducted to limit autonomous tissue mutation.
4. Core Cerebral Mapping: Examination of the individual throughout infection cycles revealed different core zones of activity, including but not limited to the neo-mammalian cortex, limbic system and stomach tissues. Further testing could improve our understanding of erratic behaviours and lead termination protocols.
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Dr. Henry Elias Nacht,
Chief Medical Supervisor, ####### Medical Bay and Scientific Institute
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